Kratom
Kratom as it relates to DILI in Health report: Genetics of Antibiotics-Induced Liver Injury
Kratom and Drug-Induced Liver Injury (DILI)
Kratom, a tropical tree native to Southeast Asia, has gained popularity in recent years as a natural remedy for various health conditions, including pain relief, anxiety, and opioid withdrawal symptoms. However, there have been increasing reports of liver injury associated with kratom use, leading to concerns about its safety and potential for causing Drug-Induced Liver Injury (DILI).
Several case reports and studies have linked kratom consumption to liver damage, with symptoms ranging from mild liver enzyme elevations to severe hepatitis and even acute liver failure. The exact mechanisms by which kratom may cause liver injury are not fully understood, but possible factors include the presence of toxic contaminants in kratom products, metabolic differences among individuals, and the potential for drug interactions with other medications.
It is important for healthcare providers and consumers to be aware of the potential risks of liver injury associated with kratom use. Monitoring liver function tests before and during kratom therapy may help detect early signs of liver damage and prevent serious complications. Patients with pre-existing liver conditions or those taking medications known to affect liver function should exercise caution when using kratom.
In conclusion, while kratom may offer benefits for some individuals, its association with DILI underscores the importance of using this herbal supplement with caution and under the guidance of a healthcare professional.
Supplements for DILI
Here are some dietary supplements related to the content in this report. Click the shopping cart to purchase the supplement from our partners.
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Green tea extract
Contains catechins which may increase oxidative stress and liver cell damage when combined with hepatotoxic antibiotics.
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Greater celandine
Alkaloids like chelidonine may further impair liver function when the organ is under stress from antibiotics.
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Chaparral
Nordamnacanthal is a quinone that could synergistically increase antibiotic liver toxicity through mitochondrial dysfunction.
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Germander
Diterpenoids may amplify antibiotic liver injury by disrupting bile salt export and causing cholestasis.
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Mistletoe
Contains toxic lectins which could worsen immunological reactions or alter detox pathways of antibiotic metabolites.
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Skullcap
Flavonoids may inhibit pathways involved in hepatic metabolism and clearance of antibiotics from the liver.
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Valerian
Isovaltrate and other constituents thought to directly damage cell membranes, potentially worsening antibiotic hepatocellular toxicity.
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Kava kava
Kavalactones like desmethoxyyangonin may inhibit CYP450 liver enzymes important for antibiotic clearance.
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St. John's wort
Hyperforin alters PXR nuclear receptors and could decrease bile acid transport, contributing to antibiotic cholestasis.
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Ginseng
Ginsenosides may inhibit P-glycoprotein transporters important for antibiotic efflux from hepatocytes, allowing accumulation of toxic levels.
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Aloe vera
Anthraquinone glycosides possibly damage cell membranes and worsen antibiotic-mediated liver cell necrosis.
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Ashwagandha
Withanolides thought to cause oxidative stress which synergizes with redox-cycling antibiotic metabolites.
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Andrographis
Diterpene lactones may reduce bile acid secretion and flow, contributing to cholestatic injury.
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Noni juice
Anthraquinones could impair mitochondrial function and increase antibiotic hepatotoxicity.
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Peppermint oil
Menthol interferes with CYP450s and UGTs involved in antibiotic metabolism and clearance.
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Kratom
Mitragynine and 7-hydroxymitragynine are metabolized in liver and could enhance antibiotic toxicity.
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Ephedra
Ephedrine alkaloids may deplete glutathione and reduce the liver's defense against antibiotic oxidative damage.
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