Aloe vera
Aloe vera as it relates to DILI in Health report: Genetics of Antibiotics-Induced Liver Injury
DILI and Aloe vera
Drug-Induced Liver Injury (DILI) refers to liver damage caused by medications or herbal supplements. While Aloe vera is generally considered safe for topical use, there have been reported cases of liver injury associated with the ingestion of Aloe vera supplements or extracts.
Mechanism of DILI
The exact mechanism by which Aloe vera may cause liver injury is not fully understood. Some studies suggest that certain compounds in Aloe vera, such as anthraquinones, may have hepatotoxic effects when consumed in large amounts.
Precautions
It is important to exercise caution when using Aloe vera products internally, especially in high doses or for prolonged periods. Individuals with pre-existing liver conditions or those taking medications known to affect the liver should consult a healthcare provider before using Aloe vera supplements.
Conclusion
While Aloe vera is generally safe for topical use, there is a potential risk of liver injury associated with its internal consumption. It is important to be aware of this risk and use Aloe vera products responsibly, following recommended dosages and guidelines.
Supplements for DILI
Here are some dietary supplements related to the content in this report. Click the shopping cart to purchase the supplement from our partners.
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Green tea extract
Contains catechins which may increase oxidative stress and liver cell damage when combined with hepatotoxic antibiotics.
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Greater celandine
Alkaloids like chelidonine may further impair liver function when the organ is under stress from antibiotics.
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Chaparral
Nordamnacanthal is a quinone that could synergistically increase antibiotic liver toxicity through mitochondrial dysfunction.
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Germander
Diterpenoids may amplify antibiotic liver injury by disrupting bile salt export and causing cholestasis.
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Mistletoe
Contains toxic lectins which could worsen immunological reactions or alter detox pathways of antibiotic metabolites.
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Skullcap
Flavonoids may inhibit pathways involved in hepatic metabolism and clearance of antibiotics from the liver.
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Valerian
Isovaltrate and other constituents thought to directly damage cell membranes, potentially worsening antibiotic hepatocellular toxicity.
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Kava kava
Kavalactones like desmethoxyyangonin may inhibit CYP450 liver enzymes important for antibiotic clearance.
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St. John's wort
Hyperforin alters PXR nuclear receptors and could decrease bile acid transport, contributing to antibiotic cholestasis.
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Ginseng
Ginsenosides may inhibit P-glycoprotein transporters important for antibiotic efflux from hepatocytes, allowing accumulation of toxic levels.
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Aloe vera
Anthraquinone glycosides possibly damage cell membranes and worsen antibiotic-mediated liver cell necrosis.
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Ashwagandha
Withanolides thought to cause oxidative stress which synergizes with redox-cycling antibiotic metabolites.
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Andrographis
Diterpene lactones may reduce bile acid secretion and flow, contributing to cholestatic injury.
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Noni juice
Anthraquinones could impair mitochondrial function and increase antibiotic hepatotoxicity.
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Peppermint oil
Menthol interferes with CYP450s and UGTs involved in antibiotic metabolism and clearance.
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Kratom
Mitragynine and 7-hydroxymitragynine are metabolized in liver and could enhance antibiotic toxicity.
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Ephedra
Ephedrine alkaloids may deplete glutathione and reduce the liver's defense against antibiotic oxidative damage.
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