Kratom
Kratom as it relates to DILI in Health report: Genetics of Antibiotics-Induced Liver Injury
Kratom and Drug-Induced Liver Injury (DILI)
Kratom, a tropical tree native to Southeast Asia, has gained popularity in the United States as an herbal supplement used for its stimulant and opioid-like effects. However, there have been increasing reports of liver injury associated with kratom use, raising concerns about its safety.
Drug-induced liver injury (DILI) is a potential risk of using kratom. DILI refers to liver damage caused by medications, supplements, or other substances. Symptoms of DILI can range from mild to severe and may include jaundice, fatigue, abdominal pain, and nausea.
Several case reports and studies have linked kratom use to liver injury. The exact mechanism by which kratom may cause liver damage is not fully understood, but it is believed to be related to the metabolism of kratom compounds in the liver.
It is important for individuals using kratom to be aware of the potential risk of liver injury and to monitor for any signs or symptoms of DILI. If any concerning symptoms develop, it is crucial to seek medical attention promptly.
In conclusion, while kratom may have potential benefits, it is essential to use it cautiously and be aware of the potential risk of liver injury, especially in the context of DILI.
Supplements for DILI
Here are some dietary supplements related to the content in this report. Click the shopping cart to purchase the supplement from our partners.
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Green tea extract
Contains catechins which may increase oxidative stress and liver cell damage when combined with hepatotoxic antibiotics.
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Greater celandine
Alkaloids like chelidonine may further impair liver function when the organ is under stress from antibiotics.
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Chaparral
Nordamnacanthal is a quinone that could synergistically increase antibiotic liver toxicity through mitochondrial dysfunction.
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Germander
Diterpenoids may amplify antibiotic liver injury by disrupting bile salt export and causing cholestasis.
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Mistletoe
Contains toxic lectins which could worsen immunological reactions or alter detox pathways of antibiotic metabolites.
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Skullcap
Flavonoids may inhibit pathways involved in hepatic metabolism and clearance of antibiotics from the liver.
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Valerian
Isovaltrate and other constituents thought to directly damage cell membranes, potentially worsening antibiotic hepatocellular toxicity.
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Kava kava
Kavalactones like desmethoxyyangonin may inhibit CYP450 liver enzymes important for antibiotic clearance.
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St. John's wort
Hyperforin alters PXR nuclear receptors and could decrease bile acid transport, contributing to antibiotic cholestasis.
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Ginseng
Ginsenosides may inhibit P-glycoprotein transporters important for antibiotic efflux from hepatocytes, allowing accumulation of toxic levels.
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Aloe vera
Anthraquinone glycosides possibly damage cell membranes and worsen antibiotic-mediated liver cell necrosis.
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Ashwagandha
Withanolides thought to cause oxidative stress which synergizes with redox-cycling antibiotic metabolites.
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Andrographis
Diterpene lactones may reduce bile acid secretion and flow, contributing to cholestatic injury.
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Noni juice
Anthraquinones could impair mitochondrial function and increase antibiotic hepatotoxicity.
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Peppermint oil
Menthol interferes with CYP450s and UGTs involved in antibiotic metabolism and clearance.
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Kratom
Mitragynine and 7-hydroxymitragynine are metabolized in liver and could enhance antibiotic toxicity.
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Ephedra
Ephedrine alkaloids may deplete glutathione and reduce the liver's defense against antibiotic oxidative damage.
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