Kava kava
Kava kava as it relates to DILI in Health report: Genetics of Antibiotics-Induced Liver Injury
Causes of DILI with Kava Kava
The exact mechanism by which kava kava causes liver injury is not fully understood. Some studies suggest that certain compounds in kava may be toxic to the liver cells, leading to inflammation and damage. Additionally, factors such as the method of preparation, dosage, and individual susceptibility may also play a role in the development of DILI.
Symptoms of DILI with Kava Kava
Symptoms of DILI associated with kava kava can vary widely and may include jaundice (yellowing of the skin and eyes), fatigue, abdominal pain, nausea, and dark urine. In severe cases, liver failure can occur, which may be life-threatening.
Prevention and Management
To reduce the risk of DILI with kava kava, it is important to use the herb cautiously and under the guidance of a healthcare provider. It is recommended to avoid excessive or prolonged use of kava, as well as to choose reputable sources for purchasing kava products. If any symptoms of liver injury develop while using kava, it is crucial to seek medical attention promptly.
In conclusion, while kava kava has been traditionally used for its calming effects, it is important to be aware of the potential risk of liver injury associated with its use. Practicing caution and monitoring for any signs of liver damage can help mitigate the risk of DILI with kava kava.
Supplements for DILI
Here are some dietary supplements related to the content in this report. Click the shopping cart to purchase the supplement from our partners.
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Green tea extract
Contains catechins which may increase oxidative stress and liver cell damage when combined with hepatotoxic antibiotics.
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Greater celandine
Alkaloids like chelidonine may further impair liver function when the organ is under stress from antibiotics.
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Chaparral
Nordamnacanthal is a quinone that could synergistically increase antibiotic liver toxicity through mitochondrial dysfunction.
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Germander
Diterpenoids may amplify antibiotic liver injury by disrupting bile salt export and causing cholestasis.
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Mistletoe
Contains toxic lectins which could worsen immunological reactions or alter detox pathways of antibiotic metabolites.
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Skullcap
Flavonoids may inhibit pathways involved in hepatic metabolism and clearance of antibiotics from the liver.
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Valerian
Isovaltrate and other constituents thought to directly damage cell membranes, potentially worsening antibiotic hepatocellular toxicity.
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Kava kava
Kavalactones like desmethoxyyangonin may inhibit CYP450 liver enzymes important for antibiotic clearance.
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St. John's wort
Hyperforin alters PXR nuclear receptors and could decrease bile acid transport, contributing to antibiotic cholestasis.
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Ginseng
Ginsenosides may inhibit P-glycoprotein transporters important for antibiotic efflux from hepatocytes, allowing accumulation of toxic levels.
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Aloe vera
Anthraquinone glycosides possibly damage cell membranes and worsen antibiotic-mediated liver cell necrosis.
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Ashwagandha
Withanolides thought to cause oxidative stress which synergizes with redox-cycling antibiotic metabolites.
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Andrographis
Diterpene lactones may reduce bile acid secretion and flow, contributing to cholestatic injury.
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Noni juice
Anthraquinones could impair mitochondrial function and increase antibiotic hepatotoxicity.
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Peppermint oil
Menthol interferes with CYP450s and UGTs involved in antibiotic metabolism and clearance.
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Kratom
Mitragynine and 7-hydroxymitragynine are metabolized in liver and could enhance antibiotic toxicity.
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Ephedra
Ephedrine alkaloids may deplete glutathione and reduce the liver's defense against antibiotic oxidative damage.
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